Help Yourself to Health

Insulin Resistance Pt 1

Three Angels Broadcasting Network

Program transcript

Participants: Agatha Thrash

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Series Code: HYTH

Program Code: HYTH000224


00:01 Hello, it was about 2000 years ago that the
00:06 ancients recognized that there were some
00:08 people, not everybody, but some people had
00:11 sweet urine. They recognized this because
00:14 if the urine spilled on the ground the ants
00:18 were attracted to it. They called the disease
00:21 as diabetes mellitus. Diabetes because that's
00:24 the Greek word for fountain and mellitus
00:27 because that's the Latin word for sweet or
00:30 honey. So, its honey fountain or we call it
00:34 diabetes mellitus or sugar disease. So, if
00:41 you would like to learn something about this
00:43 sugar disease we will be talking about this
00:46 during this program and we hope you'll join us.
01:09 Welcome to Help Yourself to Health with
01:12 Dr. Agatha Thrash of Uchee Pines Institute.
01:15 And, now here is your host, Dr. Thrash.
01:20 Now, diabetes is actually two separate
01:24 diseases. One, we call it type I diabetes and the
01:29 other we call type II diabetes. Those we
01:32 used to say, we used to call them juvenile
01:35 diabetes and the adult onset diabetes. Now,
01:39 we don't actually refer to them anymore by
01:42 those terms because we recognize now that
01:45 some adults can get that what we used to call
01:48 juvenile diabetes and now with overweight
01:52 being so prevalent among children we're
01:55 now seeing the type II diabetes very
01:57 frequently in small children. And, so we
02:01 now refer to them as type I and type II,
02:04 but there are two separate diseases and
02:06 have totally different ways, so they express
02:10 themselves in the blood test. Now, in order for
02:14 us to fully understand about this, we of course
02:18 would like to know something about how
02:21 the blood sugar is handled in the body.
02:25 And, just what we can do to make the body
02:29 better able to handle sugar. When sugar
02:33 builds up in the bloodstream that causes
02:36 an irritative problem to blood vessels. Sugar
02:40 itself is what we might call an irritant in the
02:44 blood. And insulin while it's essential to
02:49 life as sugar is also. While insulin is
02:52 essential to life, we cannot function without
02:55 it. If we get too much then we can look at it in
02:59 lay terms as an irritant. And, it causes a number
03:03 of problems for the body. One of which is
03:06 overweight that's one reason for that is, that
03:08 it's a very good appetite stimulant. So, as it goes
03:12 higher in the bloodstream, the person
03:15 has more-and-more appetite. So, if they see
03:18 something they like and they don't have habits
03:20 that are very strict and make it so that they will
03:23 not eat at certain times and will not eat certain
03:26 things that they know to be unhealthful. They
03:29 don't have those habits well ingrained then
03:32 because of the surge of appetite that comes
03:35 with the high insulin levels they tend to
03:39 overeat and to eat too frequently. And to be
03:43 willing to eat foods that are very sweet, they
03:45 tend to have a very good appetite for foods
03:49 that are very sugary or very salty or very fatty
03:53 and all of those tend to promote more appetite
03:56 and also tend to promote overweight.
03:59 So, there are some ways that we can help
04:05 ourselves by dietary restrictions, but if we
04:10 know some of the complications that can
04:12 occur from diabetes that too is a deterrent or
04:17 an inspiration or a motivating factor to
04:20 help us to learn about these diseases and to
04:23 get so that we are not ravaged by the diseases
04:28 and the complications. So, I have some pictures
04:31 to show you of some of those problems that can
04:34 occur on the skin. And the first one that I
04:38 would like to show you is a diabetic ulcer.
04:41 Now, the usual diabetic ulcer is just as you see
04:46 here with a little pus and dead material down
04:51 in the active part of the ulcer. The edges are
04:54 little raised and a little reddened and if the
04:58 usual leg will be a little different from this
05:01 because it is usually quite a lot larger and it
05:04 is quite a lot more swollen. Because of the
05:08 swelling of it, it makes the skin tense. And that
05:11 tends to draw the edges apart more and to retard
05:18 healing. So, one of the things that we can do to
05:21 improve the healing is to put on a type of
05:25 bandage that will make the, the leg, the skin on
05:32 the leg to pull together more, so that the, the
05:36 tissue can grow, can spread over, the skin
05:42 surfaces can spread over the wound and
05:45 heal it in that way. Now, another thing that
05:48 we need to do with this ulcer is to clear up the
05:51 pus and the dead material down in the
05:54 active part of the ulcer. And, there is a rigid
05:58 bandage, which we call the Unna boot, which I
06:01 would like to show to you now.
06:03 The, and for that I would like to have a
06:08 product, which you can buy on the market may
06:11 not go by this brand name, but it will have
06:14 or may have the name of Unna boot or Unna's
06:17 boot as this one does. Now, the Unna boot is
06:21 the original and of course we have used
06:24 the Unna boot for I suppose 100 years or
06:28 more and it goes on very nicely, even when
06:32 you use the old fashion Unna's boot itself. And,
06:36 I've here the ingredients for the old Unna boot
06:40 that we used to make up 50 years ago and it
06:44 is zinc oxide, which you simply measure out
06:46 into a cup and you get the formula properly
06:50 for it. And at which you can get online and then
06:54 you use glycerin. And glycerin can also be
06:57 obtained from a pharmacy as this one
07:01 was. And the glycerin is mixed with it along
07:05 with some gelatin, which you can get from
07:07 a grocery store just the plain Knox gelatin or
07:12 any kind of clear gelatin, which you can
07:15 use. I suppose Agar could also be used in
07:17 the same way, but the Knox gelatin is by all
07:21 means the best. And so you can make, you can
07:24 make this yourself, it needs to be heated
07:27 because of the gelatin, so the gelatin is
07:30 softened in some water and heated to dissolve
07:35 it and then the zinc oxide and the glycerin
07:37 put in. And then the part of foot and leg and
07:44 the ulcer is wrapped with gauze such as this
07:49 and then with a little paint brush the old
07:53 Unna paste is painted on the first layer of the
07:57 gauze and then a second layer of gauze is
08:00 wrapped on that and it's allowed to harden and
08:03 to gel and that makes the rigid bandage. Now,
08:08 while I have put on I suppose a hundred
08:12 Unna boots with the old fashion paste and the
08:16 paint brush. I would like to show you how to
08:19 use this, this new variety. This is more
08:23 expensive, but it's also it's so much more
08:26 convenient. So, I've asked an assistant and
08:30 that so Arianna Hartsfield. So, Arianna
08:33 if you will come here and join me, and I
08:35 would like you to sit right here and Arianna is
08:40 one of our students in our church school at
08:44 Uchee Pines and we are pretending that she has
08:47 a large diabetic ulcer on her left leg. So, if you
08:51 remove your left sock, we will proceed to put
08:57 on an Unna boot. Unna boots are extremely
09:00 successful and they will both clear up the ulcer.
09:06 It will clear that to dead an infective material
09:09 up. And also will make the, the swelling go
09:14 down and heal the ulcer, you can see it
09:17 from one bandage change to the next.
09:19 Okay, Arianna if you let me have your left foot
09:22 and I'll just put this right up here on my
09:24 knee that's the very best way that you can put
09:27 this Unna boot on. And then you just take the
09:32 gel cast that you have and it would be
09:35 packaged very tightly because it is, it, it
09:40 should not dry out and then you can see that
09:44 sort of sticks together little bit like this and on
09:48 the ulcer we will put a 4x4, so, let's pretend
09:53 that she has a big ulcer right here. So, we will
09:55 put this right there and then we will put this on
10:00 top of that just like so that will help it to stay
10:04 on. And, then we just wrap this on, it isn't
10:08 difficult to put it on, it goes on sort of like a
10:11 little cast. And because it is made of rather thin
10:16 gauze, it is of course going to mold a bit to
10:21 her foot and this has to go down all the way to
10:24 the base of the toes as you see me doing here.
10:28 The reason for that is so that the little rigid
10:33 bandage, once it gets rigid then it will be
10:37 right at the base of the toes and if you have to
10:40 make it fit then you can just twist it little bit as
10:43 you see me do there. And then it goes back
10:46 up and if you will notice you can see that
10:51 there is some chalky material that gets on
10:53 my fingers like that. This chalky material is
10:57 the zinc oxide, which we had in the original
11:01 Unna paste. The original Unna paste was
11:04 a very good paste with zinc oxide. One more
11:07 wrap around the foot and then it goes up over
11:11 the leg. Now, the reason that it must go
11:13 up over the leg is because the, the
11:18 swelling needs to come down, but we don't, we
11:21 don't make it tight. We simply make it fit
11:24 because the way that it's going to be
11:26 functional is that her walking and
11:31 moving and flexing her muscles will make it so
11:35 that she will have the muscles to make the
11:42 fluid go out of her leg. So, we wanted to go up
11:46 a good way on her leg just for that very
11:49 purpose. Now, each roll around leaves about
11:55 one-third of the last roll exposed. So, that its,
12:02 it's not, don't make one edge go to the next
12:06 edge just make it about like this. Now, once it
12:11 gets on before they do too much walking on it.
12:15 It has to harden and that takes with this
12:20 commercial form, they will often tell you how
12:23 long it's going to take, but it will often take
12:27 maybe a couple of hours before it gets
12:29 fully hardened, but it's not a big problem just
12:34 make it, make it fit and let them have the time
12:39 to have it to gel. Now, that looks as if it's
12:43 about enough. So, cut this off right here.
12:46 Now, you can put the rest of it back in the
12:49 water tight package and use this for the next
12:53 time you put the bandage on. Now, at
12:56 this point we need to assume a couple of
12:59 things. One is that couple of days have
13:03 gone by and it's time for a dressing change.
13:07 So, with the dressing change the way to do
13:10 that, you will notice now that this looks like
13:12 a cast, looks like the cast, so that the
13:16 orthopedists put on for a broken bone. And
13:19 that's good too, you can give it a little bit of a
13:21 rub, so that makes it smooth, but when you
13:25 take it off you simply cut it off away from the
13:29 place, where the ulcer is. Ulcer is over here
13:32 and you simply cut it off somewhere away
13:36 from the, the ulcer. So, as I'm doing now, takes
13:42 a little to do although it will be once they have
13:45 walked on it for a couple of days and
13:48 usually the dressing change is every 2-5
13:51 days depending the stage, where the ulcer
13:55 is. But let say this one, this one is ready to
13:59 come off and this is simply the way that you
14:02 do it. And sometimes the patient can, can
14:06 help you to get, get it cut off, but as you can
14:10 see it's takes a little bit of, of working to get it
14:14 off. Very affective treatment, I like it very
14:19 much for the diabetic ulcers and I'm sure that
14:23 anyone who has experienced with this
14:26 will tell you the same thing that I've had.
14:29 Now, usually after a couple of days of
14:34 having the, the bandage on, the person, the
14:38 bandage will be a little bit loose on the leg, but
14:42 the leg will be much smaller then it had been
14:45 when you put the bandage on. Thank you,
14:47 Arianna. We appreciate your working with us,
14:52 with this. That's fine.
14:53 So, little bit messy. Now, as you can see
14:58 it's, while it's a little trouble to put the
15:01 bandage on. I can assure you that it's quite
15:04 affective for this problem. Now, I have
15:08 some other pictures that I would like to show
15:10 you of other problems that can occur in the
15:14 diabetic. So, at this point we will take, we
15:18 will look at a toe. Now, here you can see that
15:20 this toe has a chronic ulcer on it. This toe
15:26 may progress in the diabetic with an ulcer
15:29 like that to amputation either the toe itself has
15:34 to be amputated or sometimes a portion of
15:38 the foot perhaps half of the foot or even a
15:41 below the knee amputation may have to
15:44 occur for such an ulcer is this. This ulcer
15:48 happened because the, a heating pad was used
15:53 for the patient and that got too hot and this
15:57 little ulcer develop. little blister first, and
16:01 then the ulcer, which would not heal and that
16:04 points out the fact that diabetics should not
16:07 have hot things put on the feet. They can't
16:11 experience the pain like a normal person can
16:14 and therefore they've serious problems with
16:18 excessive heat. Hot foot baths are best avoided.
16:22 A warm foot bath body temperature is about the
16:26 hottest that a diabetic should take a hot foot
16:29 bath or a foot bath not hot foot bath. Now, we
16:33 have another picture to show you. This picture
16:35 also shows a typical problem with a diabetic
16:39 on the just behind the great toe will develop
16:45 an ulcer on the, on the sole of the foot. It's not
16:49 an uncommon place. One of the reasons why
16:52 it develops there is that the, the diabetic cannot
16:55 see on the bottom and often because of weight
16:59 they may not be able to reach down and, and
17:02 recognize that something is
17:04 developing different on the bottom of the foot.
17:07 And because the nerves are not sensitive
17:11 anymore then the person develops an
17:15 ulcer before they know it and it's a serious
17:18 ulcer before they even know it. Now, the next
17:21 picture that I will show you is also in a
17:23 diabetic, also illustrates a typical problem. This
17:27 problem is Gangrene you will see that
17:30 developing in the right foot the second toe. See
17:35 the end of that toe is black that is called dry
17:39 Gangrene, but you will notice that on the left
17:43 toes there are, there are three of the left toes
17:46 that have the purplish red discoloration. If
17:50 you were to press your thumb into that area of
17:54 the purplish red discoloration, it might
17:57 take as long as a minute for the blood to run
18:02 back into the skin at that point. Where as a
18:05 normal person will have the, if you press
18:07 on the foot on a toe the, the normal person will
18:13 have the blood to run back into the skin in
18:17 maybe one or two seconds doesn't take
18:19 long at all. So, you can see by that, that the
18:22 diabetic is having a serious problem with
18:26 the skin. Now, let's talk a bit
18:29 about how the diabetic develops an infection
18:34 more frequently and more easily than the
18:37 average person does. Because of the fact that
18:41 when the blood sugar rises after a meal, the
18:45 bodies first objective is to get the, get all of the
18:53 sugar that's excess above the fasting level
18:56 to get all of that, out of the bloodstream and put
19:00 into various places, where sugar can be
19:04 stored. Now, sugar can be stored in muscles, it
19:08 can be stored in the kidney, in the liver, and
19:11 in the skin and various other places. But the
19:14 skin is an important immediate reservoir for
19:19 sugar. So, sugar is taken out of the
19:23 bloodstream and temporarily stored in
19:26 the skin. That makes the sugar in the skin go
19:29 high. Sugar is a good culture medium for
19:33 bacteria. So, a small scratch, a little
19:36 abrasion, can result in quite a serious
19:40 infection. And I would like to show you some
19:44 charts that I have that will illustrate this the
19:47 way that infection occurs. Let, let's
19:51 take a look here at a germ itself. This is a
19:56 bacterium one with flagella, which are little
20:04 threads with which the bacterium swims, so it
20:09 moves these and propel themselves along. Also
20:14 you will notice this on the outside is the
20:16 capsule, then comes the cell wall and then
20:20 comes the cell membrane before you
20:22 ever get to the cytoplasm then the
20:25 nucleus is down here. You can see by that,
20:28 that the germ is well armored to invade and
20:34 has good resistance against us. But, we also
20:37 have very good advancing substances
20:44 and advancing structure such as our white blood
20:47 cells and the vast array of chemical substances
20:51 that the immune system can produce. An
20:55 amazing array of substances that are
20:58 powerful to kill bacteria or to wall them off and
21:02 make them, so that they cannot move. So
21:05 bacterium maybe one without flagella, they
21:09 don't all have flagella most of them in fact do
21:12 not, but as something happens let us say,
21:16 there you get an infection somewhere on
21:20 the skin or in the internal organs and the
21:24 germ gets into the bloodstream and goes
21:28 to other parts of the body. You can get a
21:31 meningitis from, from one, from the germ
21:36 called Neisseria meningitidis and that is
21:39 the, the germ most likely to affect the
21:44 meninges then you can get a streptococcus that
21:47 can infect the number of places that can get in
21:50 the bloodstream, can even go to the heart
21:52 valves. You can get streptococcus
21:55 pneumonia, which can go to the lungs and a
21:58 streptococcus aureus, which can involve the
22:02 skin and the pseudomonas, which
22:04 can involve the prostate and the salmonella,
22:07 which can involve that the colon of course and
22:10 we are familiar with that from reports that
22:14 we have out of people, who have eaten
22:16 infected meat or eggs or chicken and this has
22:20 caused them to get a salmonella infection
22:23 in the colon. So, of course with these germs
22:28 that can go all over the body, one can certainly
22:31 see how these germs could either originate in
22:34 the skin or could go from the blood into the
22:37 skin. Now, let's take a moment for an anatomy
22:42 lesson, if you will indulge me. This is a
22:44 most wonderful illustration of the grand
22:49 design of the human body. We have in bone
22:53 marrow and before we are born these cells,
22:58 which is called here hemocytoblast or a
23:01 stem cell, you may have heard of stem cell
23:04 transplants. Well, here is a stem cell, we will say
23:08 that this is a bone marrow and this is the
23:11 red bone marrow and this is the cortical bone.
23:14 The red bone marrow such as in ribs and in
23:17 the breast bone that kind or and other flat
23:21 bones, the skull, the hip bones. These are, this
23:26 red bone marrow is the place where these
23:29 grow. Then over here we have the line that
23:33 will form red blood cells. And here is a
23:35 mature red blood cell right here in this area.
23:40 Then we have what is called the granulocytes
23:43 and they develop from a stem cell, which can
23:46 develop in anyone of our blood cells. The
23:48 stem cell, this is a myeloblast and then
23:51 we have over here the neutrophils, the
23:53 eosinophils and the basophils. Eosinophils
23:58 are increased in number when we have allergies,
24:02 intestinal parasites very common thing to cause
24:07 a very high eosinophil count. Certain collagen
24:12 diseases can also cause a high eosinophil count.
24:16 The neutrophils are our first line of attacking
24:21 soldiers. They attack any germ that comes
24:26 into the body, any bacterium that comes into
24:28 the body and will also move into any kind of
24:32 inflammatory position. Then the basophils, we
24:36 don't know as much as we would certainly like
24:38 to know about basophils, but one of
24:41 the things that we have recognized in the
24:43 basophil line is that, when there is a cancer
24:46 in the body somewhere basophils can increase
24:51 in number. Then here are the monocytes.
24:54 These are also called macrophages they can,
24:58 they can also be called dendritic cells or a
25:01 macrophages, very excellent fighting cells
25:05 for us. All of these are good fighting cells.
25:08 Then the lymphocytes and one of there line
25:11 can make antibodies and plasma cells, which
25:16 we used to think didn't have any good reason
25:20 for being. Now, we know that they are the
25:22 part of the immune system and very
25:23 important for us. Then way over here we have
25:27 what is called a megakaryoblast. And
25:30 the megakaryocyte, which develops from
25:33 the megakaryoblast has also platelets. These
25:37 platelets have a very important part in our
25:42 clotting mechanism and in abnormal... in times
25:47 when there are abnormalities in the
25:49 body, an inflammation. These platelets can
25:52 become sticky and then they make a clot. If we
25:56 get a cut they can become sticky and
25:59 make a clot. Now, in the urinary tract, we
26:04 have some other problems that can be
26:07 very common in diabetics. So, now I
26:09 would like to just show you here is the adrenal,
26:12 here is the kidney, here is the cut surface of a
26:16 normal kidney and here is a dilation of the
26:20 kidney pelvis because of a, of an impaction of
26:26 a stone that has developed here in the
26:29 kidney pelvis and the top part of the ureters,
26:31 so this the ureter gets blocked by a stone and
26:35 we've a dilation of the ureter there. Here the
26:40 ureter comes down and empties into the bladder
26:43 and the bladder can have in the diabetic
26:47 may have cystitis or may even get a little
26:51 ulceration. Here we have a jack stone type
26:57 of stone in the bladder, which is not too
27:01 uncommon. And here we've stones in the
27:06 kidney. These stones, these kinds of stones,
27:10 but then this stone is called a staghorn
27:15 calculus. Now, staghorn calculus is
27:18 quite to find for the pathologist and I have
27:22 several in my collection of stones from various
27:26 parts of the body. Stones that require
27:29 a special license for prospecting for them
27:32 and so I try to hang on to them, they
27:35 are now called semi- precious stones.
27:38 Diabetics are more likely to produce stones
27:41 then others and I hope that by our going into
27:45 these things you can understand how
27:48 diabetics must protect themselves in many
27:50 ways, but by following a good lifestyle they
27:54 too can live normally.


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Revised 2014-12-17